SpectraCell Blog

SpectraCell's Nutritional Correlation Chart on Autism

Posted by SpectraCell Laboratories, Inc. on Wed, Feb 06, 2013 @ 01:54 PM

Autism Nutritional WheelVitamin D - High dose vitamin D therapy reversed autistic behaviors in severely deficient children; Maternal vitamin D deficiency may predispose children to autism.3,4,5

Vitamin A - One cause of autism may be a defect in a retinoid receptor protein (G-alpha protein) which is critical for language processing, attention and sensory perception; Evidence suggests natural vitamin A fixes this protein defect in autistics.1,2

Folate - Oral folate therapy can resolve symptoms of autism in some cases, particularly in autistics with genes that impair folate dependent enzymes.31,32,33

Glutamine - Blood levels of this amino acid which acts as a neurotransmitter are particularly low in autistics. Glutamine also helps prevent leaky gut syndrome, which can exacerbate autistic symptoms.28,29,30

Vitamin C - Improved symptom severity and sensory motor scores in autistic patients possibly due to interaction with dopamine synthesis; Vitamin C also has a strong sparing effect on glutathione.26,27

Glutathione & Cysteine -  Commonly deficient in autistic patients, lack of these antioxidants impair detoxification and methylation processes; Low levels linked to neurological symptoms in autism which is often considered an oxidative stress disorder.21,22,23,24,25

Vitamin B1 - Deficiency linked to delayed language development; Supplementation may benefit autistic patients.19,20

Vitamin B12 - Low B12 impairs methylation (detoxification) which causes the neurological damage responsible for many autistic symptoms; Deficiency of B12 can cause optic neuropathy and vision loss in autistics; B12 raises cysteine and glutathione levels.16,17,18

Vitamin B6 - Cofactor the neurotransmitters serotonin and dopamine; Conversion of B6 to its active form is compromised in many autistics; Supplementation trials with B6 resulted in better eye contact, speech and fewer self-stimulatory behavior in autistics; Some consider B6 in combination with magnesium to be a breakthrough treatment for autism.14,15

Magnesium - Cofactor for the neurotransmitters that affect social reactions and emotion; Autistics have low levels; Improves effectiveness of B6 therapy.11,12,13

Zinc - Eliminates toxic mercury from brain tissue; Zinc/ copper ratio is particularly low in autistic kids; Low zinc impairs  the protein (called metallothionein) that removes heavy metals from the body.8,9,10

Carnitine - Transports fatty acids into cells; Low carnitine (common in autism) impairs the ability to use fatty acids for learning and social development.6,7

 For a copy of Spectracell's Nutrition Correlation chart on Autism click here

Topics: micronutrients, micronutrient testing, Cysteine, Antidepressants, zinc, folate, Vitamin D, Carnitine, Magnesium, Vitamin C, Vitamin A, Vitamin B6, B Vitamins, Antioxidants, diagnostic tools, supplements, autism, Vitamins, DNA, deficiencies, health, deficiency, Depression, Glutamine, Neurology, Glutathione, Diet, Minerals, Digestion, Brain, Nervous System, Neurotransmitters, Aggression, Stress, Vitamin B1, degenerative illness, micronutrient test

Clinical Applications of MTHFR Genomic Testing

Posted by SpectraCell Laboratories, Inc. on Tue, Jan 22, 2013 @ 03:50 PM

Clinical Applications of MTHFR Genomic Testing webinar presented by Dr. Bridget Briggs
 
What you learned:  Dr. Briggs, MTHFR
  • Methylation processes in the body. 
  • The function of MTHFR and the common polymorphisms found in 40% of Americans
  • The links between the common polymorphisms in MTHFR and risks involving cardiovascular disease, thromboembolic disease, irritable bowel, depression, memory agility, autoimmunity, poor detoxification, infertility, PMS, insomnia and many other disorders. 
  • Treatment for patients with common polymorphisms including L-5 MTHF, Methylcobalamin, B6, Trimethylglycine, inositol, and other key nutrients.

  • Case Study Review

Clinical Applications of MTHFR Genomic Testing Webinar

 

Topics: SpectraCell, Autoimmunity, cancer cells, autoimmune diseases, Telomere testing, B Vitamins, Fibromyalgia, ApoE Genotyping, Cardiovascular Health, diagnostic tools, MTHFR Genotyping, Genotyping, Multivitamins, Chronic Disease, immune system, breast cancer, deficiencies, Elderly, chronic fatigue and nutrition, health, Case Study, Inflammatory Bowel Disease, degenerative illness, Cancer Prevention, Heart Health, Gastrointestinal Tract, cardiovascular disease, Energy, Methylation, Immunidex, Immunity, eczema and nutrition, Genetics, Bowel Disease, Controversy, Dr. Bridget Briggs, Women's Health

Vitamins, minerals and antioxidants can help!

Posted by SpectraCell Laboratories, Inc. on Thu, Jan 10, 2013 @ 01:20 PM

Is carnitine the answer for male infertility?male, infertility
A group of men (n=96) who had been diagnosed as infertile for at least 18 months were given the following nutritional formulation daily for four months: L-carnitine, acetyl-L-carnitine, fructose, citric acid, selenium, coenzyme Q10, zinc, vitamin C, vitamin B12 and folic acid (see abstract for exact dosages).  At the end of the study, sperm motility improved and 16 of the patients had achieved pregnancy.  The authors concluded that carnitine may be the key component of the supplement cocktail for improving sperm quality. (Italian Archives of Urology and Andrology, September 2012)

LINK to ABSTRACT Prospective open-label study on the efficacy and tolerability of a combination of nutritional supplements in primary infertile patients with idiopathic astenoteratozoospermia.

 

Vitamin D helps leg ulcers heal
In this double-blind, placebo controlled trial, 26 patients Vitamins, Vitamin Dwith leg ulcers were given either placebo or 50,000 IU vitamin D weekly for two months.  Leg ulcer size, blood levels of vitamin D and pain was measured before and after the two month trial.  In the vitamin D group, leg ulcers were reduced in size by 28% while the placebo group had only a 9% reduction in ulcer size. The authors stated “there was a trend toward better healing in those with vitamin D reposition.” (Journal of Brazilian College of Surgeons, October 2012)

LINK to ABSTRACT Vitamin D and skin repair: a prospective, double-blind and placebo controlled study in the healing of leg ulcers.
LINK to FREE FULL TEXT

 

Complexity of methylation reactions gains insightmethyl donor, nutrients
This review emphasizes how methyl donor nutrients such as choline, folic acid and methionine interact and how consumption (via supplement or food) of one can have sparing effect s on another – such as choline’s  sparing effect on methionine, for example. (Current Opinion in Clinical Nutrition and Metabolic Care, January 2013)

LINK to ABSTRACT The nutritional burden of methylation reactions.
LINK to FLYER on NUTRIENT INTERACTIONS in METHYLATION

For more journal articles by disease or nutrient please click here

 

Topics: SpectraCell, serine, micronutrients, Coenzyme Q10, Oleic Acid, Cysteine, autoimmune diseases, zinc, Vitamin D, Carnitine, Magnesium, Choline, Vitamin C, Vitamin E, Vitamin A, Selenium, Vitamin B6, B Vitamins, Folic Acid, Antioxidants, lipoprotein particle profile, Omega 3 Fatty Acid, diagnostic tools, vitamin, wellness, pregnancy, Serum, Vitamin K, Vitamin B12, supplements, Multivitamins, Nutrition, diabetes, immune system, E-zinc, N-acetylcysteine, DNA, Calcium, Fertility, Lipoic Acid, deficiencies, health, Case Study, Omega 3s, Depression, Glutamine, Minerals, Neurotransmitters, Stress, Vitamin B1, micronutrient test, Vitamin B5, Vitamin B2, Nutritional Deficiency, Vitamin B3, cardiovascular disease, Hormones, Reproductive Health, Chromium, Manganese, Muscle recovery, Erectile Dysfunction, infertility, Niacin, Prostate, Energy, Methylation, Carbohydrate Metabolism

The Importance of Vitamins

Posted by SpectraCell Laboratories, Inc. on Fri, Jan 04, 2013 @ 01:12 PM

New study says multivitamins just don’t cut it when it comes to preventing heart diseaseVitamins, multi-vitamins

In the landmark Physician’s Health Study II, authors concluded that taking a multivitamin for over a decade did nothing to prevent cardiovascular events such as myocardial infarction or stroke. The study monitored 14,641 male doctors for over eleven years who took either a daily multivitamin or placebo and no differences in cardiovascular events or mortality was found between the two groups.  Since evidence linking deficiencies to heart disease is strong (see vitamin D study below on 45,000 patients), some conclude that a multivitamin is simply not effective in correcting deficiencies and that targeted supplementation for the individual is a better approach. (Journal of the American Medical Association, November 2012)
(American Journal of Cardiology, October 2010)

Link to ABSTRACT Multivitamins in the prevention of cardiovascular disease in men: the Physicians' Health Study II randomized controlled trial.

Link to ABSTRACT Relation of vitamin D deficiency to cardiovascular risk factors, disease status, and incident events in a general healthcare population.

Vitamin C reduces fatigue and perception of effort after exercise

Vitamins, vitamin cIn this interesting study on twenty obese adults, each were given either 500 mg of vitamin C or placebo daily for four weeks.  Their diet was strictly controlled for vitamin C content and their heart rates and fatigue scores as well as subjective perceptions of exertion were measured after exercise.  Those taking vitamin C had lower fatigue scores and those on placebo had higher fatigue scores. Heart rates and “ratings of perceived exertion” were also improved in the vitamin C group. (Nutrition, January 2013)   

Link to ABSTRACT Vitamin C status and perception of effort during exercise in obese adults adhering to a calorie-reduced diet.

For more articles and information, click here for the complete library on clinical updates.

Topics: Coenzyme Q10, zinc, Vitamin D, Vitamin C, Vitamin E, Vitamin A, Vitamin B6, B Vitamins, Folic Acid, Copper, diagnostic tools, Heart Disease, vitamin, Vitamin K, Vitamin B12, supplements, Multivitamins, E-zinc, Vitamins, deficiencies, Heart Attack, Diet, Minerals, Vitamin B1, micronutrient test, Vitamin B5, High Blood Pressure, Vitamin B2, Heart Health, Vitamin B3, Aging, Stroke

Can Nutrition Cure Depression?

Posted by SpectraCell Laboratories, Inc. on Fri, Dec 21, 2012 @ 11:09 AM

Presented by: Mary Ann Block, D.O.Depression, sad, nutrition

Topics of Discussion:

    • How Depression is Often a Symptom of Underlying Medical Problems
    • Nutrients Depleted by Antidepressants
    • Role of Zinc in the Development and Treatment of Mood Disorders
    • Case Study Review

 

Download our archived webinar presentation, "Can Nutrition Cure Depression?"

 

Also, check our webinar library for a complete list of previous webinars on a wide range of topics.

 

Topics: micronutrient testing, Antidepressants, autoimmune diseases, zinc, Vitamin D, Carnitine, Magnesium, Vitamin B6, B Vitamins, Antioxidants, Fatigue, Cholesterol, diagnostic tools, wellness, Vitamin B12, Multivitamins, Chronic Disease, E-zinc, deficiencies, chronic fatigue and nutrition, inositol, health, Case Study, website, Depression, Diet, Digestion, micronutrient test, Vitamin B2, Vitamin B3, Energy, Bipolar, webinar, Blood Pressure, Amino Acid, caffeine, Women's Health

SpectraCell's Nutritional Correlation Chart on Diabetes

Posted by Char Perez on Mon, Dec 03, 2012 @ 10:23 AM

Micronutrients such as niacin, magnesium, calcium, zinc, carnitine, inositol, alpha-lipoic acid, as well as vitamins E, B6 and D all play an important role in the prevention and treatment of diabetes.

Though diabetes is a serious disease - with the right treatment - living a longer, healthier life can be made easier.

THE ROLE OF MICRONUTRIENTS IN DIABETIC HEALTH

Vitamin E - Confers protection against diabetes by protecting pancreatic B-cells from nutrition reference chart for diabetesoxidativestress induced damage; May prevent progression of type I diabetes.

Vitamin D - Lowers risk of type I and 2 diabetes; Suppresses inflammation of pancreatic B-cells. Vitamin D receptor gene linked to diabetes.

Vitamin B3 - Preserves B-cell function in type I diabetics; Part of GTF (glucose tolerance factor) which facilitates insulin binding.

Vitamin B12 - Deficiency common in diabetics because metformin depletes B12.

Chromium - Helps insulin attach to cell's receptors increasing glucose uptake into cell; Deficiency can cause insulin resistance; Supplementation trials show dose-dependent benefits for type II diabetics.

Biotin - Stimulates glucose-induced insulin secretion in pancreatic B-cells; High dose biotin can improve glycemic control in diabetics.

Magnesium - Deficiency reduces insulin sensitivity; Low magnesium exacerbates foot ulcers in diabetics.

Zinc - Needed in the synthesis, storage and secretion of insulin; Protects pancreatic B-cells from damage; Affects the expression of genes linked to diabetes.

Lipoic Acid - Enhances glucose uptake in skeletal muscle tissue; Improves glucose tolerance in type 2 diabetics; very effective treatment for diabetic neuropathy.

Glutathione & Cysteine - Glutathione-containing enzymes protect B-cells which are particularly sensitive to oxidative stress; Type 2 diabetics have abnormal antioxidant status; Supplementation with the glutathione precursor cysteine restores antioxidant status.

Coenzyme Q10 - Protects kidney from diabetes related damage; Improves glycemic control in type 2 diabetics.

Glutamine - Stimulates a hormone called GLP-I (glucagon-like peptide I) that regulates insulin secretion after meals; Improves insulin signaling and sensitivity.

Carnitine - Reduces and even prevents pain from diabetic neuropathy; Improves insulin sensitivity by increasing glucose uptake and storage.

Inositol - Evidence suggests that inositol may be effective in treating diabetic neuropathy.

Vitamin C - Lowers glycolysated hemoglobin (HbA1c) and fasting and post-meal glucose levels and in type 2 diabetics.  

To assess a copy of the flyer which illustrates the interaction of micronutrients and their effect on patients' diabetes click here:  http://www.spectracell.com/media/disease-wheel-diabetes.pdf

Topics: SpectraCell, micronutrient testing, Coenzyme Q10, Cysteine, zinc, Vitamin D, Carnitine, Magnesium, Vitamin C, Vitamin E, B Vitamins, Vitamin B12, diabetes, immune system, Lipoic Acid, biotin, inositol, deficiency, Glutamine, Glutathione, micronutrient test, Wound Healing, Vitamin B3, reference chart, Chromium, Insulin

The Importance of Nutrition on Weight Loss

Posted by SpectraCell Laboratories, Inc. on Mon, Jun 04, 2012 @ 11:57 AM

Micronutrient TestingBelow is a list of various nutrients that affect a person's ability to gain or lose weight.

  • Zinc - reduces leptin, a beneficial hormone that regulates appetite, which is reversed by zinc repletion.
  • Asparagine - this amino acid increases insulin sensitivity which helps the body store energy in muscle instead of storing it as body fat.
  • Biotin - boosts metabolism by improving glycemic control (stabilizes blood sugar) and lowering insulin, a hormone that promotes fat formation.
  • Carnitine - carries fatty acids into cell so they can be burned for fuel; Helps reduce visceral adiposity (belly fat).
  • Calcium - inhibits the formation of fat cells; Also helps oxidize (burn) fat cells.
  • Lipoic Acid - improves glucose uptake into cells, which helps a person burn carbohydrates more efficiently.
  • Chromium - makes the body more sensitive to insulin, helping to reduce body fat and increase lean muscle.
  • Vitamin B5 - taking B5 lowers body weight by activating lipoprotein lipase, an enzyme that burns fat cells. One study linked B5 supplementation to less hunger when dieting.
  • Magnesium - low magnesium in cells impairs a person’s ability to use glucose for fuel, instead storing it as fat; Correcting a magnesium deficiency stimulates metabolism by increasing insulin sensitivity. Magnesium may also inhibit fat absorption.
  • Glutamine - reduces fat mass by improving glucose uptake into muscle.
  • Cysteine - supplementation with this antioxidant reduced body fat in obese patients.
  • Inositol - supplementation may increase adiponectin levels.
  • Vitamin B3 (niacin) - treatment with B3 increases adiponectin, a weight-loss hormone secreted by fat cells; Niacin-bound chromium supplements helped reduced body weight in clinical trials.
  • Vitamin A - enhances expression of genes that reduce a person’s tendency to store food as fat; Reduces the size of fat cells.
  • Vitamin E - inhibits pre-fat cells from changing into mature fat cells, thus reducing body fat.
  • Vitamin D - deficiency strongly linked to poor metabolism of carbohydrates; Genes that are regulated by vitamin D may alter the way fat cells form in some people.
  • Vitamin K - poor vitamin K status linked to excess fat tissue; Vitamin K helps metabolize sugars.

Download our 1-page flyer which illustrates the information above, HERE!

Weight Loss Document

Also, learn more about micronutrient testing and the importance of correcting vitamin deficiencies in our Clinical Education Center.

Topics: Asparagine, Cysteine, zinc, Vitamin D, Carnitine, Magnesium, Vitamin E, Vitamin A, B Vitamins, Vitamin K, Weight Loss, Calcium, Lipoic Acid, biotin, inositol, Glutamine, Chromium, Weight Gain

Nutritional Relationships of Hypothyroidism

Posted by SpectraCell Laboratories, Inc. on Wed, Feb 01, 2012 @ 10:16 AM

Nutritional RelationshipsBelow is a list of nutrients which significantly affect Hypothyroidism:

  • Glutathione - Hypothyroidism decreases efficacy of some antioxidants, such as glutathione peroxidase and superoxide dismutase
  • B Vitamins - A deficiency in B6, B12 or B9 (folate) can cause elevated homocysteine, which is linked with hypothyroidism. Folic acid levels have been linked to levels of thyroid stimulating hormone (TSH).
  • Vitamin C and E - Partially restores thyroid function when liver detoxification ability is compromised.
  • Vitamin A - Activates gene that regulates TSH (thyroid stimulating hormone)
  • Zinc - Increases thyroid hormone T3 in deficient subjects.
  • Copper - Low levels seen in experimentally induced hypothyroidism; Indirectly affects thyroid status by its antioxidant role via superoxide dismutase.
  • Selenium - Converts thyroid hormone T4 (thyroxine) into T3 (triiodothyronine); Deficiency reduces T3 levels causing classic hypothyroidism symptoms such as fatigue, depression or weight gain.
  • Asparagine - This amino acid is part of the structure of thyroid stimulating hormone which regulates communication with other hormones.
  • Carnitine - Decreased tissue levels of carnitine in both hypo- and hyperthyroidism contribute to muscle fatigue.
  • Lipoic Acid - Improves endothelial function in people with subclinical hypothyroidism; Protects thyroid cells from oxidative stress; May interfere with T4 therapy
  • Choline - Hypothyroidism negatively affects choline function in the brain, which can affect mood and cognition.


Topics: Asparagine, zinc, Carnitine, Choline, Vitamin C, Vitamin E, Vitamin A, Selenium, B Vitamins, Copper, Lipoic Acid, Glutathione, Hypothyroidism

A New Twist on a Common Supplement: Using Omega 3’s to Lower Homocysteine

Posted by SpectraCell Laboratories, Inc. on Wed, Jan 25, 2012 @ 09:54 AM

Omega 3's and HomocysteinGuest Blog by: Dr. Arland Hill (D.C.)

Homocysteine continues to gain acceptance as a risk factor in a number of conditions.  Once thought to be associated with mostly cardiovascular disease, homocysteine is now recognized as a contributor to numerous states of dysfunction, including arthritis, cognitive decline, osteoporosis and many more.  Given the vast reaching effects of homocysteine, having an ideal treatment protocol in place to address elevations seems necessary, if not even absolutely crucial.

Examination of the literature to lower homocysteine can be confusing at times, with some studies showing lack of efficacy of B vitamins.  However, these studies were not without criticism and ultimately a return to the basics of physiology has shown B vitamins to be an efficacious intervention.  When looking at the methionine cycle and delving deeper into the biochemistry of homocysteine, it becomes rather clear that without methyl donors, B Vitaminshomocysteine conversion to methionine gets stalled and homocysteine begins to build up.  As such, the methyl donors B12 and folate have become the primary interventions for homocysteine lowering therapy.

For some however, B12 and folate therapy are not enough.  Most of these individuals fall under the category of having a variant for the methyltetrahydrofolate reductase enzyme, commonly abbreviated MTHFR.  Variants in this enzyme impair the ability to reduce folate, thus making the conversion of homocysteine to methionine inefficient and promoting accumulation.  This is seemingly overcome fairly easily in elevated cases by the use of L 5-methyltetrahydrofolate.  Upon normalization of homocysteine in this manner, levels can generally be kept in an ideal range by the use of folate, a non-synthetic, as compared to folic acid.

As relatively straightforward as this all seems, the story does not end here.  A deeper look at the influences on our genes shows the complexity of modulating homocysteine.  There must exist a constant balance between homocysteine and methionine.  If the objective is to lower homocysteine, downregulating the enzymes that lead to its conversion and upregulating those that convert it to methionine would appear ideal.  To do this very thing, one should look no further than omega 3’s.  The polyunsaturated omega 3’s, known to most of us as fish oils, canOmega 3s directly affect the expression of the genes that control the enzymes linked to homocysteine metabolism.  Since the omega 3’s do not directly insert into the biochemical pathway of the methionine cycle, their actions clearly have to be based on another mechanism of action, one we now recognize as genetic.

Consider the therapeutic potential here for a number of patients.  Let’s take the aforementioned conditions for which homocysteine is known to affect.  Equally validated in the literature to support cardiovascular disease, arthritis, cognitive decline, and osteoporosis, omega 3’s will not only reduce inflammation but will also modify genetic expression of those enzymes metabolizing homocysteine.  This makes testing and adding omega 3’s to any clinical protocol at minimum a consideration given the considerable effects of homocysteine and the therapeutic potential of omega 3’s.

Please review the website below for more information: http://www.ncbi.nlm.nih.gov/pubmed/22260268

Dr. Arland HillArland Hill, DC, MPH, DACBN- Complete Care Chiropractic and Wellness

For more information about our client Dr. Hill, please visit his website or his blog. Or contact him at 281-557-7200.

Topics: Homocysteine, B Vitamins, supplements, Omega 3s, Dr. Arland Hill

Can Nutrient Deficiencies Provide Insight into the Health of a Neuron and Your Brain?

Posted by SpectraCell Laboratories, Inc. on Mon, Jan 09, 2012 @ 10:55 AM

Nutritional Brain HealthGuest Blog by: Dr. Arland Hill (D.C.)

Most of us are familiar with the saying use it or lose it.  Many times when individuals say this, they are likely referring to muscle tissue.  However, this saying is equally valid regarding the health of the brain and nervous tissue.  Since the brain has impact on every system in the body, keeping it healthy is of the utmost importance.  Doing so takes 3 key ingredients; energy, fats, and stimulation.

While most probably don’t think about the energy demands of the nervous system, they are in fact quite high.  The production of energy for a neuron, or nerve cell, goes beyond just good function.  The ability to produce energy is the difference between life and death.  As with all cells, an energy substrate must be available, preferably glucose.  Uptake of glucose by neurons depends on healthy insulin receptors.  Healthy insulin receptors that are sensitive optimize neuron function by efficiently controlling synapse density, promoting neuronal growth, or neuroplasticity, and refining the function of the involved neurocircuitry.  In short, neurons function better and extend their network when insulin receptors are sensitive.  But as attractive as this sounds, it fails to happen when nutrient deficiencies such as chromium and B3 exist.  Chromium and niacin help make up the glucose tolerance factor which has significant bearing on the glucose-insulin interaction.

Neuron Once glucose is taken in by the neuron, it must enter the mitochondria to produce ATP, the energy molecule.  However, to get ATP, several key nutrients must be available.  These nutrients fuel each step of the energy cycle and include B1, B2, B3, B5, glutamine, and magnesium just to name a few.  For the neuron, the importance of this cannot be understated.

When the neuron is able to receive adequate oxygen, it can combine the metabolites formed from the energy production cycle and generate ATP.  In the absence of adequate nutrient stores, ATP is not produced efficiently leading to the demise of the neuron.  This can be the early onset of neurodegeneration.  Moreover, since neurons depend on stimulation from each other to maintain functional neurocircuitry, losing a neuron will in turn have effects on adjacent neurons.  The potential “snowball-like” effect of neurodegeneration emphasizes the consequence of allowing seemingly harmless nutrient deficiencies to persist.

Lastly, a protective coating is needed.  Think about this as insulation for the wiring of your neurocircuity.  With it, neurological impulses are transmitted at a faster rate.  Our insulation is fat.  Micronutrient assessment provides a window into how we might be producing insulation Neuronsaround our neurological tissue.  Not only does such testing show how fats are being utilized by living cells, it also illustrates the status of nutrients such as B12 that are equally needed for production of our insulation, otherwise known as myelin.

The health of the nervous system is a commonly overlooked.  When it is functioning appropriately, it is given little attention.  In contrast, by the time a neurological condition manifests, it is difficult to make up lost ground to neurodegeneration.  However, altering its effects can take place, but only in the presence of adequate nutrient status.  Given proper precursors and stimulation, the ability of the neuron to produce energy is regained, promoting an environment of neuroplasticity.

Dr. Arland Hill

 

Arland Hill, DC, MPH, DACBN- Complete Care Chiropractic and Wellness

For more information about our client Dr. Hill, please visit his website or his blog. Or contact him at 281-557-7200.

Topics: micronutrient testing, Magnesium, B Vitamins, Glutamine, Brain, Nervous System, Chromium, Dr. Arland Hill, Glucose